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Arab Countries are lagging Behind in Telecommunications only two Arab countries, Morocco and Tunisia, are party to the february 1997 WTO agreement on telecoms liberlization. The agreement, involving 69 countries will open up 75 % of the world telecoms market next years, and much of the remainder by 2003. With most governments in the regions resisting telecoms liberalisation, Arab countries not participating in the WTO market access arrangements will continue to pay high call charges, and network modernmisation and expansion will be slow incomparison to the rapid developments elsewhere. Although telephone density is high in the Gulf, Arab countries as a whole account for less than 2% of world telephone revenue. Egypt The most populous Arab country has only 3 million telephone lines, equivalent to only five lines per 100 people, one of the lowest densities in the world outside sub-Saharan Africa. The National Telecommunications Organization Arento ; , a state monopoly that has resisted calls for privatization and liberalization, handles telephone provision. Instead of looking for private sector capital to expand and modernise the system, Egypt has relied on the United States Agency for International Development USAID ; . USAID has provided US million to extend switching systems, much of it recycled to AT&T, which currently has two major contracts worth US million. Plans for a digital mobile phone GSM system were scrapped due to security concerns, although a more limited car-phone GSM system is being introduced. However, Arento has recently relaxed its monopoly position and allowed a new digital communications experiment in 200 Egyptian village. A software-driven system, developed by Cyber Digital of New York, is beig managed by a new Egyptian venture, the National Telecommunications Company. Which is a subsidiary of the state-owned National Bank of Egypt. Although this remains second best to private provision, it does bypass the bureaucratic systems of Arento. Since TRAP either can exist a monomeric protein or as a proteolytically processed dimer, held together by a disulphide bridge, it was of interest to investigate if the proteolytically processed form was present in different organs Paper II ; . Both FPLC and Western blot showed that the proteolytically processed form is present in all organs examined. The results also suggested that cathepsin L might be responsible for the proteolytic processing of TRAP in certain cell types given that TRAP and cathepsin L were co localised in spleen, thymus, colon and kidney. As the proteolytically processed form of TRAP was present in all tissues and since proteolytic processing had been shown to increase the enzyme activity of TRAP9, 10, we investigated if proteolytic processing as such was sufficient to generate an active enzyme Paper II ; . The data obtained suggested that this was not the case. Proteolytic processing in the absence of reducers only led to a minimal reduction of the substrate affinity and no increase in enzyme activity. However, the proteolytic processing seems to be important for the reduction of the diiron centre since the enzyme activity of monomeric TRAP only is increased ~8 fold in the presence of reducers compared to ~26-90 folds for TRAP processed by cathepsin L and K. In conclusion, TRAP s from all rat organs tested exhibit the characteristic features of PAPs. Proteolytically processed TRAP was found in all organs examined and in spleen, thymus, colon and kidney cathepsin L could possibly participate in the proteolytic processing of the enzyme. The proteolytic processing does not increase the TRAP enzyme activity but seems to enable reduction of the diiron centre.

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Researchers within the Common Mental Disorders programme are currently involved in various international collaborations see Table on international collaboration ; . Several also serve international functions, some as members of editorial boards of scientific journals Prof. Beekman for Journal of Aging and Mental Health, Prof. Kerkhof for Crisis: the Journal of Suicide Prevention, and Dr. Penninx for the European Journal of Aging and the Journal of Gerontology: Medical Sciences ; , others as partners in international consortia Dr. Droes and Dr. Meiland in the European Community Concerted Action on the Epidemiology and Prevention of Dementia Group EURODEM ; , Prof. Jonker in the European Dementia Panel of the European Federation of Neurological Societies EFNS ; , and Prof. Kerkhof in the World Health Organisation's European Network on Suicide Prevention ; . In 2005, various researchers also received awards recognising their important contributions in research: Prof. Kerkhof received the Ivonne van de Ven Award, Dr. Droes and Dr. Meiland received the EMGO Societal Impact Award for their 43. Not a primary eye irritant. May cause mechanical irritation. Because of the shape of individual wollastonite particles, minor skin irritation may result from prolonged physical contact. Not a sensitizer. Not an ingestion hazard. Inhalation of high concentrations may cause mechanical irritation and discomfort. Therapy was not used in any of our studies, except for radiation to the base of the skull for patients with cranial nerve root involvement. The best timing for CNS prophylaxis is controversial. Omura et all7 introduced prophylaxis after patients achieved CR. However, others have suggested that earlier intervention is needed.33In our analysis, patients in the VAD and modifiedVAD regimens received prophylaxis high-dosesystemicchemotherapy or IT chemotherapy ; after achieving CR and little benefit was derived in high-risk patients. The hyperCVAD introduced IT chemotherapy earlyin the induction course. This early intervention appears to be most effective, because the incidence of CNS leukemia, both overall and in the high-risk population, was lowest in the hyperCVAD regimen 3% and 2%, respectively ; . Moreover, evidence of CNS leukemia appearing early during induction chemotherapy wasalmost eliminated with this approach Table 4 ; . Consequently, it is reasonable to recommend initiation of prophylaxis early during induction chemotherapy. One potential disadvantage is the contamination ofCSF with peripheral blood and blasts, which may confuse the interpretation of the results. Because of the toxicityof CNS prophylaxis, it is desirable to identify those patients who would benefit most from this therapy and minimize the exposure to those at lower risk. Several risk factors have been identified to be associated with CNS relapse in children, including high WBC counts, T-cell or B-cell disease, young age, thrombocytopenia, lymphadenopathy, hepatomegaly, or ~plenomegaly?~-~~ In adults, a multivariate analysis identified 1 ; high serum LDH levels and 2 ; high proliferative index ie, S + G2M compartment 214% ; as the most significant factors to predict for CNS leukemia?' Patients without any of these features had a 4% incidence of CNS leukemia at 1 year compared with 13% to 29% for those with one of these features and 56% for patients with both features.3o In present analysis, two our therapeutic modalities had a risk-oriented approach following this model: the modified VAD program, which used IT chemotherapy only among high-risk patients, and the hyperCVAD program, which included IT chemotherapy for both low- and high-riskpatients but for only 4doses in the former and 16 in the latter. This approach proved useful, because the incidence of CNS leukemia decreased most significantly in the high-risk group to 20% and 2%, respectively, which compares favorably to that in low-risk patients. The two programswith no differentiation between low- andhighrisk prohylaxis preVAD and VAD ; had significantlyhigher incidence in the high-risk group. fact that there has been The no statistically significant improvement with more aggressive prophylaxis in the low-risk group is probably due to an already low incidence without prophylaxis that would require a larger population to document small changes in incidence. Our studies have shown the leukemic cell proliferative index to be a significant factor predictive for risk of CNS disease. By itself, it identifies11% of patients withCNS disease ie, with a normal LDH level ; . However, because of the limited availability of this measurement, we are currently.

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TEXAS A&M UNIVERSITY. TEXAS A&M UNIVERSITY PRESS Continued ; . Z TA475.7 B735 NO.6 Trench knives and mustard gas : with the 42nd Rainbow Division in France Hugh S. Thompson ; edited, with an introduction by Robert H. Ferrell. 1st ed. c2004. xii, 205 p. C.A. Brannen series ; no. 6 ; . On the way -Somewhere in France -- Rimaucourt -- The raid -- The trenches -- More of same -- To Bru and back -- Badonviller -- The last trench days -- Paradise -- Hospital and home -- Wounded again -- Recovery -- Home again -- Preparation -- St. Mihiel -- The end. Includes bibliographical references p. [199]-200 ; and index. 0MF and phenelzine.
Renal and vascular effects of atrial natriuretic factor during cardiopulmonary bypass M Hynynen, R Palojoki, J Heinonen, I Tikkanen, AL Harjula and F Fyhrquist Chest 1991; 100; 1203-1209 DOI 10.1378 chest.100.5.1203 This information is current as of March 14, 2008. Department of Pharmacology M.S., O.P., V.G. ; , Department of Histology and Embryology Y.M. ; , and Department of Physiology A.P., M.A. ; , Faculty of Medicine in Hradec Kralove and Department of Biochemical Sciences, Faculty of Pharmacy T.S., H.K. ; , Charles University in Prague, Hradec Kralove, Czech Republic; and Lady Davis Institute for Medical Research, Departments of Physiology and Medicine, McGill University, Montreal, Quebec, Canada P.P and phenobarbital.
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Lena Marions, friend and colleague, always there with her valuable support. Christian Fiala, co-author, for all his help and friendship. My co-authors Ann-Kristin Ulfgren, Anneli Stavreus-Evers, Olof Stephansson, Fredrik Granath, Bernhard Watzer, HW Schweer who have been a pleasure to work with. A special thank to Berit Stby for all their help and support. Professor Sten Cekan for statistical analysis and kind help and support. Margareta Hellborg and Lena Elffors Sderlund, research nurses at WHO Centre for their excellent work and highly professional care of the patients and for always giving me kind support and for all the good time we shared. Monika Johansson and Eneli Salomonsson, staff at SESAM and Maijken Ullmark, Marietta Molnar, Eva-Lisa Nordlund, Marianne Racke and Inga-Maj Andersson, staff at C21 for their warm and professional care of patients and kind support. Helena von Hertzen and Paul Van Look at the WHO Centre in Geneva for their kind collaboration. The team at WHO centre, for all your valuable help and kindness. Astrid Hggblad for all her help during my years as a PhD student. Catharina Karlsson for help and friendship . Eva Eneroth I will miss you ; and other colleagues in labour ward for all the nice time we shared. Birgitta Bystrm and other colleagues at the FoU-laboratory for their valuable support during the years. Professor PC Ho, Hong Kong for sharing his ideas about sublingual administration of misoprostol. 48.

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Than 30% ; , noninducibility does not portend a good prognosis 198 ; . Persistent inducibility while receiving antiarrhythmic drugs predicts a worse prognosis 199 ; . Patients in whom amiodarone suppressed VT inducibility or slowed VT to a mean cycle length of greater than 400 ms had 30% higher mortality compared with patients who did not respond to amiodarone and had an ICD placed instead 200 ; . EP-guided antiarrhythmic drug effectiveness in patients with NSVT who had induced sustained VT conferred no benefit 195 ; . The prognostic value of inducible ventricular flutter and VF is still controversial. Limited data on the prognostic value of inducible ventricular flutter suggest that it may be an important endpoint 201, 202 ; . 5.3.2. Electrophysiological Testing in Patients With Dilated Cardiomyopathy In DCM, EP testing plays a minor role in the evaluation and management of VT. This is related to low inducibility, reproducibility of EP study, and the predictive value of induced VT 203, 204 ; see Section 9.1 ; . 5.3.3. Electrophysiological Testing in Repolarization Anomalies Due to Genetic Arrhythmia Syndromes 5.3.3.1. Long QT Syndrome EP testing has not proved useful in LQTS 103, 205 ; see Section 11.1.1 for further discussion ; . 5.3.3.2. Brugada Syndrome The role of EP testing for risk stratification in Brugada syndrome is debated 104, 206 208 ; , and it will probably remain undefined until prospective data are obtained in patients studied with a uniform protocol in a large population with adequate follow-up see Section 11.1.3 for further discussion ; . 5.3.3.3. Hypertrophic Cardiomyopathy The value of EP testing in HCM has been controversial see Section 9.2 for further discussion ; . 5.3.3.4. Arrhythmogenic Right Ventricular Cardiomyopathy The arrhythmic manifestations of ARVC are variable 96 ; . The prognostic role of EP testing in patients presenting with isolated PVCs or NSVT is not known. The response to EP testing may be influenced by the severity of the disease. Progression of disease has to be considered see Section 9.3 for further discussion ; . 5.3.4. Electrophysiological Testing in Patients With Outflow Tract Ventricular Tachycardia EP testing for the evaluation of outflow tract VT is basically similar to that for other VT entities. It is motivated by the need to establish precise diagnosis to guide curative catheter ablation 209, 210 ; see Section 12.1 for further discussion and phenylephrine.

Service Code HCPCS Code Revenue Code Description -- -- -- 00000 250 DIPHENOXYLATE HCL-ATROPINE A398 00000 250 ANTIPYRINE-OXYQUINOLINE-BENZO B398 00000 250 ANTIPYRINE-OXYQUINOLINE-BENZO A399 00000 250 NEOMYCIN-POLYMYXIN-HC B399 00000 250 NEOMYCIN-POLYMYXIN-HC A400 00000 250 NITROGLYCERIN-DEXTROSE B400 00000 250 NITROGLYCERIN-DEXTROSE B401 00000 250 TRIFLUOPERAZINE HCL C404 00000 250 IBUPROFEN B406 00000 250 LIDOCAINE DIPHENYHY MAG HYDROX C407 00000 250 MILK OF MAGNESIA CASCARA SUSP A408 00000 250 MULTIPLE VITAMINS C409 00000 250 DR SHACKS A413 00000 250 PHENAZOPYRIDINE HCL A413 00000 250 PHENAZOPYRIDINE HCL A414 00000 250 DICYCLOMINE HCL A415 00000 250 ASPIRIN-BUTALBITAL-CAFFEINE A415 00000 250 ASPIRIN-BUTALBITAL-CAFFEINE A417 00000 250 DACTINOMYCIN A418 00000 250 BISOPROLOL FUMARATE A418 00000 250 BISOPROLOL FUMARATE A419 00000 250 FLUCONAZOLE A420 00000 250 AMYLASE-LIPASE-PANCRTN-PROTEAS A438 00000 997 INFLUENZA VIRUS VACCINE A439 00000 250 OMEPRAZOLE A440 00000 250 DEXTROSE 5%-RINGERS A441 00000 250 CODEINE PHOSPHATE-ASPIRIN A442 00000 250 HYDROCODONE-ACETAMINOPHEN A4828 00000 250 DIPHTHERIA TOXOID-TETANUS TOXO Total Number of Records: Sum Total of In-Patient Price: Sum of Total of Out-Patient Price: End of Report! 882 28, 500.01.

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10. Darmaun, D, Matthews, DE, Bier, DM. 1986 Glutamine and glutamate kinetics in humans. Am.J.Physiol. 251: E117-E126. 11. De Feo P, Volpi E, Lucidi P et al. 1993 Physiological increments in plasma insulin concentrations have selective and different effects on synthesis of hepatic proteins in normal humans. Diabetes. 42: 995-1002. 12. Fryburg DA, Barrett EJ. 1993 Growth hormone acutely stimulates skeletal muscle but not wholebody protein synthesis in humans. Metabolism. 42: 1223-7. 13. Fryburg DA, Louard RJ, Gerow KE, Gelfand RA, Barrett EJ. 1992 Growth hormone stimulates skeletal muscle protein synthesis and antagonizes insulin's antiproteolytic action in humans. Diabetes. 41: 424-9. 14. Fukagawa NK, Minaker KL, Rowe JW, Goodman MN, Matthews DE, Bier DM, Young VR. 1985 Insulin-mediated reduction of whole body protein breakdown. Dose-response effects on leucine metabolism in postabsorptive men. J Clin Invest. 76: 2306-11. 15. Garcia-Estevez DA, Araujo-Vilar D, Cabezas-Cerrato J. 1998 Non-insulin-mediated glucose uptake in several insulin-resistant states in the postabsortive period. Diabetes Res Clin Pract. 39: 107-13. 16. Hansen I, Tsalikian E, Beaufrere B, Gerich J, Haymond M, Rizza R. 1986 Insulin resistance in acromegaly: defects in both hepatic and extrahepatic insulin action. J Physiol. 250: E269-73. 17. Karlander S, Vranic M, Efendic S. 1986 Increased glucose turnover and glucose cycling in acromegalic patients with normal glucose tolerance. Diabetologia 29: 778-83. 18. Kasayama S, Otsuki M, Takagi M et al. 2000 Impaired beta-cell function in the presence of reduced insulin sensitivity determines glucose tolerance status in acromegalic patients. Clin Endocrinol 52: 549-55. 19. Kimball, S, Jefferson L, Fadden P, Haystead T, and Lawrence J. 1996 Insulin and diabetes cause reciprocal changes in the association of eIF4E and PHAS-I in rat skeletal muscle. J Physiol Cell Physiol 270: C705-C709 and phenylpropanolamine.

Alexandria Area Economic Development Commission AAEDC ; Out in Front Alexandria Pro-Fab Co. Inc. Back to their Roots Alexandria, MN Out in Front Back to their Roots Alexis Bailly Vineyard Queen of the Vine Vintage Minnesota: Alexis Bailley Vineyard brings the flavor of France to the fields of Hastings Alfalfa America Inc. Alfalfa firm buys Excelsior Henderson site Alfred Brickhouse LLC AW Properties sells Edina building Alfred's Grand Petit Magasin Kim Salmela Dresses up retail empiore with Alfred's Algos Therapeutics Try, Try, Try Again Gain From Pain Alignor Negotiating Growth: Software maker Alignor looks to create win-win scenarios All Flex Inc. Where Are They Now? All Paper Recycling Inc. Beyond Bricks and Sticks All terrain vehicles ATVs ; Artic Cat sells more ATVs than snowmobiles Polaris to flaunt rugged military ATVs Allen Interactions Inc. Are We Players? It's how you say it that counts: Allen Interactions' DialogCoach brings voice recognition to training Allete Inc. Allete Q3 earnings drop Allete completes spin-off of Adesa Allete announces spinoff, stock split Allete 2003 net earnings rise 72% Alley Ventures VCs hopeful about funding as St. Paul Cos. Retreats Alliant Techsystems Inc. ATK earnings down on pension expenses ATK closes on PSI deal, ups guisdance for 2005 ATK defense contract could be worth M ATK CEO Murphy salary, bonus jump.

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Person calling animal exposures excluded ; The majority of calls about exposure to a poison were received from members of the public. Person calling Number of calls % Family member Self Doctor Nurse Friend Carer Pharmacist Ambulance Counselling service Other medical professional, eg psychologist, social worker Other, eg police, teachers Total 17200 4104 2232 and photofrin.

Species present as the appendix proceeds through the developmental stages up to and including thermogenesis Elthon and Mclntosh, 1987; Rhoads and Mclntosh, 1992 ; . As illustrated in Figure 7, the S. guttatum appendix alternative oxidase also shows a shift during development in the relative proportions of the oxidized and reduced species present, with the oxidized species being relatively more abundant before thermogenesis and the reduced species being predominant during and following thermogenesis. Alternative oxidase activity showed some correspondence with this change in proportion of oxidized and reduced species Fig. 7 and data not shown ; . However, activity levels also corresponded with the total amount of alternative oxidase protein present and appearance of the two additional lower molecular mass forms which do not resolve well in S. guttatum when DTT is omitted from the gel sample buffer. Precooling and water ingestion on thermoregulation and physical capacity during exercise in a hot environment. J Sports Sci in press ; . 19. Hensel H. Thermoreception and temperature regulation. London: Academic Press, 1981, pp. 1-321. 20. Hori T, Kiyohara T, Nakashima T, Shibata M, and Koga H. Multimodal responses of preoptic and anterior hypothalamic neurons to thermal and nonthermal homeostatic parameters. Can J Physiol Pharmacol 65: 1290-1298, 1987. Ishiwata T, Hasegawa H, Yazawa T, Otokawa M, and Aihara Y. Functional role of the preoptic area and anterior hypothalamus in thermoregulation in freely moving rats. Neurosci Lett 325: 167-170, 2002. Jones DJ, Veale WL, and Copper KE. Alterations in body temperature elicited by intrahypothalamic administration of Tetrodotoxin, ouabain and A23187 ionophore in the consicious cat. Brain Res Bull 5: 75-80, 1980. Kanosue K, Hosono T, Zhang Y-H, and Chen XM. Neuronal net works controlling thermoregulatory effectors. In: Brain Function in Hot Environment Progress in Brain Research, vol. 115 ; , edited by Sharma HS, and Westman J. Amsterdam: Elsevier, 1998, 115, p. 49-62. 24. Kay D, Taaffe DR, and Marino FE. Whole-body pre-cooling and heat storage during self-paced cycling performance in warm humid conditions. J Sports Sci 17: 937-944, 1999. Kiyohara T, Hirata M, Hori T, and Akaike N. Hypothalamic warm-sensitive neurons possess a tetrodotoxin-sensitive sodium channel with a high Q10. Neurosci Res 8: 48-53, 1990. Marino FE. Methods, advantages, and limitations of body cooling for exercise and pilocarpine.

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Dear colleagues, During the last years, the Latin American Society for Sexual and Impotence Research SLAIS ; - the Latin American branch of ISSM became a very strong and active society in Sexual Medicine in all Latin America. We gave a lot of educational courses in many different countries, increasing the number of members to more than 50%. Another landmark of our society is the high level of our last regional meetings: we achieved a high standard scientific level together with a precise and fantastic organization. And prepare yourselves because we are going to do it again this year! It's my pleasure to invite you to the SLAIS next meeting to be held in Punta del Este, Uruguay, from 1st to 3rd December 2005. The scientific program is almost ready and we'll have many experts sharing with everybody the most recent advances in Sexual Medicine. I assure you will enjoy this very exciting program! Punta del Este in an unforgettable and wonderful beach, with a very nice weather at that time of the year. And the congress venue The Conrad Resort & Casino Punta del Este - is one of the most known and beautiful resorts of all Latin America. Again we'll be joining the pleasure of working with the joy of living! Don't forget and note in your agenda: SLAIS 2005, Punta del Este, Uruguay from December 1st to 3rd, 2005. See you there and pima. 1. Ho, D. D., Neumann, A. U., Perelson, A. S., Chen, W., Leonard, J. M., Markowitz, M. 1995 ; Rapid turnover of plasma virions and CD4 lymphocytes in HIV-1 infection. Nature 373, 123126. 2. Orenstein, J. M., Fox, C., Wahl, S. M. 1997 ; Macrophages as a source of HIV during opportunistic infections. Science 276, 18571861. 3. Gorantla, S., Che, M., Gendelman, H. E. 2005 ; Isolation, propagation, and HIV-1 infection of monocyte-derived macrophages and recovery of virus from brain and cerebrospinal fluid. Methods Mol. Biol. 304, 35 48. Stout, R. D., Suttles, J. 2004 ; Functional plasticity of macrophages: reversible adaptation to changing microenvironments. J. Leukoc. Biol. 76, 509 513. Rosenberg, Z. F., Fauci, A. S. 1990 ; Immunopathogenic mechanisms of HIV infection: cytokine induction of HIV expression. Immunol. Today 11, 176 180. Fantuzzi, L., Belardelli, F., Gessani, S. 2003 ; Monocyte macrophagederived CC chemokines and their modulation by HIV-1 and cytokines: a complex network of interactions influencing viral replication and AIDS pathogenesis. J. Leukoc. Biol. 74, 719 725. Graziosi, C., Gantt, K. R., Vaccarezza, M., Demarest, J. F., Daucher, M., Saag, M. S., Shaw, G. M., Quinn, T. C., Cohen, O. J., Welbon, C. C., Pantaleo, G., Fauci, A. S. 1996 ; Kinetics of cytokine expression during primary human immunodeficiency virus type 1 infection. Proc. Natl. Acad. Sci. USA 93, 4386 4391. Poli, G., Biswas, P., Fauci, A. S. 1994 ; Interferons in the pathogenesis and treatment of human immunodeficiency virus infection. Antiviral Res. 24, 221233. 9. Bleul, C. C., Farzan, M., Choe, H., Parolin, C., Clark-Lewis, I., Sodroski, J., Springer, T. A. 1996 ; The lymphocyte chemoattractant SDF-1 is a ligand for LESTR fusin and blocks HIV-1 entry. Nature 382, 829 833. Raport, C. J., Gosling, J., Schweickart, V. L., Gray, P. W., Charo, I. F. 1996 ; Molecular cloning and functional characterization of a novel human CC chemokine receptor CCR5 ; for RANTES, MIP-1 , and MIP-1 . J. Biol. Chem. 271, 1716117166. 11. Samson, M., Labbe, O., Mollereau, C., Vassart, G., Parmentier, M. 1996 ; Molecular cloning and functional expression of a new human CC-chemokine receptor gene. Biochemistry 35, 33623367. 12. Vives, R. R., Imberty, A., Sattentau, Q. J., Lortat-Jacob, H. 2005 ; Heparan sulfate targets the HIV-1 envelope glycoprotein gp120 coreceptor binding site. J. Biol. Chem. 280, 2135321357. 13. Bobardt, M. D., Saphire, A. C., Hung, H. C., Yu, X., Van der Schueren, B., Zhang, Z., David, G., Gallay, P. A. 2003 ; Syndecan captures, protects, and transmits HIV to T lymphocytes. Immunity 18, 2739. 14. Turville, S. G., Vermeire, K., Balzarini, J., Schols, D. 2005 ; Sugarbinding proteins potently inhibit dendritic cell human immunodeficiency virus type 1 HIV-1 ; infection and dendritic-cell-directed HIV-1 transfer. J. Virol. 79, 13519 13527. Chehimi, J., Luo, Q., Azzoni, L., Shawver, L., Ngoubilly, N., June, R., Jerandi, G., Farabaugh, M., Montaner, L. J. 2003 ; HIV-1 transmission and cytokine-induced expression of DC-SIGN in human monocyte-derived macrophages. J. Leukoc. Biol. 74, 757763. 16. Geijtenbeek, T. B., Kwon, D. S., Torensma, R., van Vliet, S. J., van Duijnhoven, G. C., Middel, J., Cornelissen, I. L., Nottet, H. S., KewalRamani, V. N., Littman, D. R., Figdor, C. G., van Kooyk, Y. 2000 ; DC-SIGN, a dendritic cell-specific HIV-1-binding protein that enhances trans-infection of T cells. Cell 100, 587597. 17. Turville, S., Wilkinson, J., Cameron, P., Dable, J., Cunningham, A. L. 2003 ; The role of dendritic cell C-type lectin receptors in HIV pathogenesis. J. Leukoc. Biol. 74, 710 718.

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